Level 1 of 6Core
Tachyarrhythmias
The core to-do list — diagnose and manage, at a glance
Diagnose— recognise it
- Classify in three answers: any rhythm above 100/min — stable or unstable, narrow or broad, regular or irregular; those three answers give the treatment before the name does.
- Any one adverse feature mandates electricity, not drugs: shock (systolic BP < 90 mmHg, cold peripheries, capillary refill > 3 s, confusion), syncope, new ischaemic chest pain or ST change, heart failure, or rate > 150/min with poor perfusion. No palpable central pulse = cardiac arrest — unsynchronised defibrillation.
- Broad QRS (≥ 0.12 s) is VT until proven otherwise: approximately 80% of broad-complex tachycardias are VT, more still with previous infarction — in all cases of doubt diagnose and treat as VT. A normal blood pressure does not exclude VT.
- A regular narrow tachycardia at almost exactly 150/min is atrial flutter with 2:1 block until proven otherwise — sawtooth F waves at about 300/min, revealed but not terminated by adenosine.
- Three rhythms carry the highest immediate lethality: VT with haemodynamic compromise; torsades de pointes (complexes twisting about the baseline, QTc > 0.50 s); and pre-excited atrial fibrillation — irregular, very broad, very fast (often > 200/min) with beat-to-beat variation in QRS width, which can degenerate into ventricular fibrillation.
- Bedside bloods: potassium (aim 4.0–4.5 mmol/L in ventricular arrhythmia), magnesium, glucose, troponin, digoxin level if taking it, and thyroid function — mandatory in unexplained new atrial fibrillation. In infants true SVT runs above 220/min and is monotonously unvarying.Not available at your setup — Serum electrolytes.
Manage now— do this, in order
- Unstable, any rhythm — synchronised DC cardioversion: sedate the conscious patient, and confirm the synchronisation markers before *every* shock — most defibrillators revert to unsynchronised mode after each discharge. Paediatric energy 1 J/kg first shock, then 2 J/kg.Doctor / NurseNot available at your setup — Defibrillator.
- If three synchronised shocks fail: amiodarone 300 mg IV over 10–20 minutes, repeat the shock, then amiodarone 900 mg over 24 hours; correct potassium and magnesium and try an anteroposterior pad position.Doctor / NurseNot available at your setup — Defibrillator.
- Stable, narrow, regular — vagal manoeuvres first (terminate 20–50%): modified Valsalva — semi-recumbent at 45°, blow into a 10 mL syringe for at least 15 seconds, then lie flat with legs raised to 45° for 15 seconds. Infants: iced flannel over forehead, eyes and nose bridge for up to 15 seconds.
- Adenosine is the first-line drug (terminates AVNRT/AVRT in ~90%): 6 mg rapid IV push over 1–2 seconds with an immediate 20 mL sodium chloride 0.9% flush into the largest proximal vein; if no effect in 1–2 minutes, 12 mg, then a further 12 mg. Start at 3 mg with a central line, dipyridamole or a transplanted heart. Paediatric: 0.1 mg/kg (max 6 mg), then 0.2 mg/kg, then 0.3 mg/kg (max 12 mg). A defibrillator must be to hand — it provokes AF in up to 12%.Doctor / NurseNot available at your setup — Defibrillator.
- If adenosine fails, choose ONE AV-nodal blocking class and stay within it: verapamil 5–10 mg IV over 5–10 min, or diltiazem 0.25 mg/kg IV over 2 min, or metoprolol 5 mg IV every 5 min (max 15 mg). Never verapamil with a broad QRS, never verapamil with a beta-blocker (or the reverse), and verapamil is contraindicated in infants under 1 year.Doctor / Nurse
- Stable, broad, regular — treat as VT: amiodarone 300 mg IV over 20–60 minutes then 900 mg over 24 hours, diluted in 5% dextrose through the largest vein (paediatric 5 mg/kg over 20–60 minutes); or lidocaine 1 mg/kg IV bolus then 1–4 mg/min. One antiarrhythmic at a time — if the drug fails, sedate and cardiovert rather than adding a second.Doctor / Nurse
- Torsades de pointes: magnesium sulphate 2 g (8 mmol) IV over 10–15 minutes in every case (paediatric 25–50 mg/kg, max 2 g); stop every QT-prolonging drug; correct potassium to 4.0–4.5 mmol/L; raise the rate with overdrive pacing at 90–120/min or isoprenaline 2–20 micrograms/min. Never give amiodarone, sotalol or any QT-prolonging antiarrhythmic in torsades.Doctor / Nurse
- Pre-excited AF: synchronised DC cardioversion is the treatment of choice. Verapamil, diltiazem, digoxin, beta-blockers and adenosine are absolutely contraindicated — they block the protective AV node and precipitate ventricular fibrillation. If genuinely stable and a drug is needed: procainamide 10–17 mg/kg at 20–50 mg/min, or ibutilide 1 mg over 10 minutes repeated once.Doctor / Nurse
- Stable AF — rate control first: metoprolol where there is ischaemia, diltiazem where blood pressure is low, never both classes IV together; targets < 110/min without symptoms, < 80/min if symptoms persist. Cardioversion without anticoagulation only if AF < 48 hours (Davidson's: 24 hours — prefer the safer figure); otherwise anticoagulate 3 weeks before or exclude thrombus by transoesophageal echo, and continue at least 4 weeks after. Correct potassium, magnesium, hypoxia and acidosis in parallel — nothing works in an uncorrected patient.Doctor / Nurse
Synchronised cardioversion — initial biphasic energies
| Rhythm | Energy |
|---|---|
| Regular narrow SVT | 100 J (70–120 J), escalating |
| Atrial flutter | 100–200 J |
| Atrial fibrillation | 200 J → 360 J / machine max |
| Pre-excited AF | Treat as AF — high energy from the outset |
| VT with a pulse | 100–200 J, escalating |
| Paediatric, any rhythm | 1 J/kg, then 2 J/kg |
Refer / escalate
Escalate or transfer urgently: any tachyarrhythmia with an adverse feature (shock, syncope, ischaemia, heart failure, rate > 150/min with poor perfusion), any broad-complex tachycardia, torsades de pointes, pre-excited AF, anyone needing cardioversion, amiodarone infusion, pacing or a defibrillator, and any new AF needing echocardiography, thyroid assessment and an anticoagulation decision.
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